Author

Antonio M. Estévez

Consejo Superior de Investigaciones CientíficasORCID 0000-0002-8272-5405Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Guillermo PerisiD· 2 shared Lourdes López-OnievaiD· 2 shared Pablo Tristán-RamosiD· 2 shared Sara R. HerasiD· 2 shared Alasdair C. IvensiD· 1 shared Amador GallardoiD· 1 shared Ana Gázquez-GutiérreziD· 1 shared Antonio Sánchez–PozoiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersMolecular Biology

Guillermo PerisORCID

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2 papers

Recent·Most cited

Molecular BiologyNucleic Acids ResearchPeer-reviewedFeb 20, 2026

Control of retrotransposon-driven activation of the interferon response by the double-stranded RNA binding protein DGCR8

Ana Gázquez-Gutiérrez, Priscilla Chin, Guillermo Peris…·Sara Macías

This study shows DGCR8 suppresses endogenous dsRNA from TEs to prevent IFN response activation, with implications for 22qDS.

2 citations

humanbulk RNA-seq

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Molecular BiologybioRxivPreprintJun 18, 2024

BMAL1 represses transposable elements independently of CLOCK in pluripotent cells

Amador Gallardo, Efres Belmonte‐Reche, María Marti-Marimon…·David Landeira

This study shows BMAL1 represses transposable elements in pluripotent cells independently of CLOCK, leading to genome-wide changes and totipotency features when BMAL1 is deleted.

DOI 10.1101/2024.06.18.599568

mouseDNA methylation

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