Author

Priscilla Chin

University of Edinburgh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Molecular Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Alasdair C. IvensiD· 2 shared Ana Gázquez-GutiérreziD· 2 shared Guillermo PerisiD· 2 shared Katrina GordoniD· 2 shared Lisanne I KnoliD· 2 shared Pablo Tristán-RamosiD· 2 shared Sara MacíasiD· 2 shared Sara R. HerasiD· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersMolecular Biology

Alasdair C. IvensORCID

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Author profile →

3 papers

Recent·Most cited

Intestinal bacteria hijack an evolutionarily conserved epithelial repair signal

Sam Benson, Priscilla Chin, Sebastian Rogatti, Alessandro Scopelliti, Luigi Zechini, Rosalind Heron, Charlotte Dawson, Anna E. Davey, Mariska S. Simpson, Amanda O. Wong, Jair Marques, Alex von Kriegsheim, David Harold Dockrell, Christopher D. Lucas, Jenna L. Cash, , Christopher J. Anderson

The paper identifies hypoxanthine as a key metabolite released during apoptosis that is consumed by gut bacteria, impairing epithelial repair.

bioRxiv · Aug 11, 2026 · PreprintMicrobiology

Control of retrotransposon-driven activation of the interferon response by the double-stranded RNA binding protein DGCR8

Ana Gázquez-Gutiérrez, Priscilla Chin, Guillermo Peris, Katrina Gordon, Pilar G. Marchante, Jeroen Witteveldt, Pablo Tristán-Ramos, Jérôme O. Rouvière, Lisanne I Knol, Alasdair C. Ivens, Lourdes López-Onieva, Antonio M. Estévez, William Garland, Torben Heick Jensen, Sara R. Heras, Sara Macías

This study shows DGCR8 suppresses endogenous dsRNA from TEs to prevent IFN response activation, with implications for 22qDS.

Nucleic Acids Research · Feb 20, 2026Peer-reviewedMolecular Biology

DGCR8 haploinsufficiency leads to primate-specific RNA dysregulation and pluripotency defects

Ana Colomer-Boronat, Lisanne I Knol, Guillermo Peris, Laura Sánchez, Silvia Peluso, Pablo Tristán-Ramos, Ana Gázquez-Gutiérrez, Priscilla Chin, Katrina Gordon, Guillermo Barturen, Robert E. Hill, Francisco Jose Sanchez-Luque, Jose Luis Garcia-Perez, Alasdair C. Ivens, Sara Macías, Sara R. Heras

This study shows that DGCR8 haploinsufficiency in human pluripotent cells causes RNA dysregulation and pluripotency defects, with rescue by primate-specific miRNAs.

Nucleic Acids Research · Mar 20, 2025Peer-reviewedMolecular Biology