Author

Danny Ben‐Zvi

Hebrew University of JerusalemORCID 0000-0002-8697-9744Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:benchmark study· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Hadar Arien‐ZakayiD· 1 shared Haya Benhayon· 1 shared James B. KoprichiD· 1 shared Jochen RoeperiD· 1 shared Jonathan M. BrotchieiD· 1 shared Joshua A. GoldbergiD· 1 shared Lora KovachevaiD· 1 shared Menachem Hanani AiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Hadar Arien‐ZakayORCID

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2 papers

Recent·Most cited

Benchmarking single-cell foundation models reveals an advantage in the low-data regime for cell-type and disease-state prediction

Haya Benhayon, Michael M. Danziger, Danny Ben‐Zvi, Michal Rosen‐Zvi

The paper benchmarks single-cell foundation models for cell-type and disease-state prediction, finding they outperform baselines in low-data regimes.

bioRxiv · Oct 12, 2026 · PreprintBioinformatics

α-Synuclein-induced Kv4 channelopathy in mouse vagal motoneurons causes non-motor parkinsonian symptoms

Wei‐Hua Chiu, Lora Kovacheva, Ruth E. Musgrove, Hadar Arien‐Zakay, James B. Koprich, Jonathan M. Brotchie, Rami Yaka, Danny Ben‐Zvi, Menachem Hanani A, Jochen Roeper, Joshua A. Goldberg

This study identifies a Kv4 channelopathy in mouse vagal motoneurons caused by α-synuclein overexpression, leading to impaired gastrointestinal motility as a non-motor prodromal symptom of Parkinson's disease.

Science Advances · Mar 10, 2021Peer-reviewedNeuroscience