Author

Erin A. Mack

University of FloridaORCID 0000-0002-8893-0792Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Organism:E. coli· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

David R. AllrediD· 2 shared Yu-Ping Xiao· 2 shared Christopher M ThomasiD· 1 shared Clinton A ElgiD· 1 shared David SneddoniD· 1 shared Elizabeth Soderling· 1 shared Eva M TopiD· 1 shared Jack SullivaniD· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersMolecular Biology

David R. AllredORCID

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3 papers

Recent·Most cited

Evolution of a Plasmid Regulatory Circuit Ameliorates Plasmid Fitness Cost

Clinton A Elg, Erin Mack, Michael Rolfsmeier, Thomas C McLean, David Sneddon, Olivia Kosterlitz, Elizabeth Soderling, Solana Narum, Paul A Rowley, Jack Sullivan, Christopher M Thomas, Eva M Top

This study shows that inactivating the upf31 gene in a plasmid improves its maintenance in E. coli by altering gene regulation, demonstrating how plasmid evolution can affect host range.

bioRxiv · Feb 5, 2024Microbiology

Babesia bovis Rad51 ortholog influences switching of ves genes but is not essential for segmental gene conversion in antigenic variation

Erin A. Mack, Massimiliano S. Tagliamonte, Yu-Ping Xiao, Samantha Quesada, David R. Allred

This paper investigates the role of BbRad51 in segmental gene conversion (SGC) and antigenic variation in Babesia bovis, finding that while BbRad51 influences transcriptional switching and SGC tract lengths, it is not essential for SGC itself.

PLoS Pathogens · Aug 31, 2020Peer-reviewedMolecular Biology

Knockout of Babesia bovis rad51 ortholog and its complementation by expression from the BbACc3 artificial chromosome platform

Erin A. Mack, Yu-Ping Xiao, David R. Allred

This paper knocks out the Bbrad51 gene in Babesia bovis and shows that an artificial chromosome platform can complement the loss of function, though it may cause recombination with native chromosomes.

PLoS ONE · Aug 6, 2019Peer-reviewedMolecular Biology