Author

Karolina Gronkowska

University of ŁódźORCID 0000-0002-9121-3700Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Organism:human· 3 Approach:statistical model· 3 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Agnieszka RobaszkiewicziD· 3 shared Sylwia MichlewskaiD· 3 shared Kinga Kołacz-MilewskaiD· 2 shared Adrianna Stępień· 1 shared Andrzej Kazimierz BednarekiD· 1 shared Eleonore Froehlich· 1 shared Maciej BorowieciD· 1 shared Magdalena StrachowskaiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersCancer Biology

Agnieszka RobaszkiewiczORCID

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3 papers

Recent·Most cited

Txn-Txnrd1 system supports redox rewiring during polyaneuploid transition and protects giant cancer cells at new redox homeostasis

Kinga Kołacz-Milewska, Karolina Gronkowska, Sylwia Michlewska, Markus Absenger, Eleonore Froehlich, Agnieszka Robaszkiewicz

The paper identifies the Txn-Txnrd1 system as critical for redox homeostasis in polyaneuploid giant cancer cells, linking it to therapy resistance and poor patient outcomes.

bioRxiv · Aug 31, 2026 · PreprintCancer Biology

BRG1 targeting overcomes ABCC-based multidrug resistance induced by paclitaxel

Karolina Gronkowska, Sylwia Michlewska, Tomasz Płoszaj, Magdalena Strachowska, Adrianna Stępień, Maciej Borowiec, Andrzej Kazimierz Bednarek, Agnieszka Robaszkiewicz

This study shows that BRG1-based SWI/SNF complex promotes ABCC gene transcription, contributing to paclitaxel resistance, and that targeting BRG1 can overcome this resistance.

Journal of Pharmacology and Experimental Therapeutics · Nov 10, 2025Peer-reviewedCancer Biology

P53 supresses transcription of the p300-E2F1-dependent gene subset by maintaining KDM5B associated with gene promoters

Karolina Gronkowska, Kinga Kołacz-Milewska, Sylwia Michlewska, Agnieszka Robaszkiewicz

This study shows p53 suppresses E2F1-driven genes by maintaining KDM5B at promoters, inhibiting p300-mediated transcription in response to cisplatin.

bioRxiv · Aug 25, 2025 · PreprintMolecular Biology