Author

Kinga Kołacz-Milewska

University of ŁódźORCID 0000-0002-2400-9371Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Agnieszka RobaszkiewicziD· 2 shared Karolina GronkowskaiD· 2 shared Sylwia MichlewskaiD· 2 shared Eleonore Froehlich· 1 shared Markus Absenger· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

5 top collaborators · 10 mutual co-author links

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2 shared papersCancer Biology

Agnieszka RobaszkiewiczORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintAug 31, 2026

Txn-Txnrd1 system supports redox rewiring during polyaneuploid transition and protects giant cancer cells at new redox homeostasis

Kinga Kołacz-Milewska, Karolina Gronkowska, Sylwia Michlewska…·Agnieszka Robaszkiewicz

The paper identifies the Txn-Txnrd1 system as critical for redox homeostasis in polyaneuploid giant cancer cells, linking it to therapy resistance and poor patient outcomes.

DOI 10.64898/2026.08.27.746985

humanbulk RNA-seq

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Molecular BiologybioRxivPreprintAug 25, 2025

P53 supresses transcription of the p300-E2F1-dependent gene subset by maintaining KDM5B associated with gene promoters

Karolina Gronkowska, Kinga Kołacz-Milewska, Sylwia Michlewska, Agnieszka Robaszkiewicz

This study shows p53 suppresses E2F1-driven genes by maintaining KDM5B at promoters, inhibiting p300-mediated transcription in response to cisplatin.

DOI 10.1101/2025.08.25.672089

humanbulk RNA-seq

𝕏 PostBluesky