Victoria A. Lukashkina

Connexin 30 mutation rescues high-frequency hearing, reduces gap junctional coupling and alters potassium currents in cochlear Deiters cells

This paper investigates how a Connexin 30 mutation affects cochlear electrophysiology, showing it rescues high-frequency hearing loss while impairing low-frequency hearing through changes in gap junction coupling and potassium currents in Deiters cells.

Optogenetics reveals roles for supporting cells in force transmission to and from outer hair cells in the mouse cochlea

The paper uses optogenetics to study the role of supporting cells in force transmission to and from outer hair cells in the mouse cochlea, finding that hyperpolarizing these cells suppresses cochlear amplification and alters mechanical responses.

A gap-junction mutation in the mouse cochlea reveals cochlear amplification is driven by outer hair cell extracellular receptor potentials

This study shows that a gap-junction mutation in mice preserves high-frequency hearing by maintaining extracellular receptor potentials in outer hair cells, suggesting these potentials drive cochlear amplification at high frequencies.