Author

Mohamed-Bilal Fares

École Polytechnique Fédérale de LausanneORCID 0000-0002-9243-0336Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:benchmarking· 1 Organism:human· 1 Assay:protein structure· 1 Assay:proteomics· 1

Frequent Co-Authors

Hilal Ahmed LashueliD· 2 shared Anass ChikiiD· 1 shared Erik StoopsiD· 1 shared Francesco Simone RuggeriiD· 1 shared Giovanni DietleriD· 1 shared Lara Petricca· 1 shared Layane Hanna-El-Daher· 1 shared Lorène Aeschbach· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Hilal Ahmed LashuelORCID

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2 papers

Recent·Most cited

Comparative analysis of total alpha-synuclein (αSYN) immunoassays reveals that they do not capture the diversity of modified αSYN proteoforms

Lara Petricca, Nour Chiki, Layane Hanna-El-Daher, Lorène Aeschbach, Ritwik Burai, Erik Stoops, Mohamed-Bilal Fares, Hilal Ahmed Lashuel

The paper compares three commercial alpha-synuclein immunoassays and finds they fail to capture the diversity of modified alpha-synuclein proteoforms.

Journal of Parkinson s Disease · May 3, 2022Peer-reviewedNeuroscience

N-terminal phosphorylation of Huntingtin: A molecular switch for regulating Htt aggregation, helical conformation, internalization and nuclear targeting.

Sean M. DeGuire, Francesco Simone Ruggeri, Mohamed-Bilal Fares, Anass Chiki, Urszula Cendrowska, Giovanni Dietler, Hilal Ahmed Lashuel

This study investigates how phosphorylation at S13 and S16 of Httex1 affects its structure, aggregation, and subcellular localization, revealing a phosphorylation-dependent switch that regulates its function.

Journal of Biological Chemistry · Sep 5, 2018Peer-reviewedNeuroscience