Author

Shivakumar R. Veerabhadraiah

University of UtahORCID 0000-0002-9353-4591Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:mouse· 3 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Michael J. JuryneciD· 3 shared Matthew Honeggar· 2 shared Nikolas H. KazmersiD· 2 shared Abhijit SukuliD· 1 shared Anna E. Miller· 1 shared Antonio C. Zelada· 1 shared Anupama BinoyiD· 1 shared Birgit SchillingiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersGenetics

Michael J. JurynecORCID

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3 papers

Recent·Most cited

Sirt5 regulates chondrocyte metabolism and osteoarthritis development through protein lysine malonylation

Huanhuan Liu, Anupama Binoy, Siqi Ren, T. C. Martino, Anna E. Miller, Craig R. G. Willis, Shivakumar R. Veerabhadraiah, Abhijit Sukul, Joanna Bons, Jacob Rose, Birgit Schilling, Michael J. Jurynec, Shouan Zhu

This study identifies Sirt5's role in regulating chondrocyte metabolism and osteoarthritis through lysine malonylation, showing its dysregulation contributes to OA in aging and obesity.

Arthritis & Rheumatology · Apr 3, 2025Peer-reviewedCell Biology

Tenascin C Deletion Impairs Tendon Healing and Functional Recovery After Rotator Cuff Repair

Robert Z. Tashjian, Jared L. Zitnay, Nikolas H. Kazmers, Shivakumar R. Veerabhadraiah, Antonio C. Zelada, Matthew Honeggar, Matthew C. P. Smith, Peter Nissen Chalmers, Heath B. Henninger, Michael J. Jurynec

This study investigates the role of Tenascin C in tendon healing after rotator cuff repair in mice, finding impaired healing and altered gene expression in TNC-deficient mice.

Journal of Orthopaedic Research® · Nov 27, 2024Peer-reviewedGenetics

The NOD/RIPK2 signaling pathway contributes to osteoarthritis susceptibility

Michael J. Jurynec, Catherine M. Gavile, Matthew Honeggar, Ying Ma, Shivakumar R. Veerabhadraiah, Kendra A. Novak, Kazuyuki Hoshijima, Nikolas H. Kazmers, David Jonah Grunwald

The paper identifies the NOD/RIPK2 signaling pathway as a contributor to osteoarthritis susceptibility through genetic and mouse model studies.

bioRxiv · Feb 7, 2022 · PreprintGenetics