Ikuko Fujiwara

The paper investigates how Spiroplasma MreB isoforms deform membranes, showing that SpeMreB5 drives deformation while SpeMreB4 suppresses it, with nucleotide state affecting the process.

This study uses TIRF microscopy and crystal structures to show Cytochalasin D caps and severs actin filaments, with distinct binding modes and concentrations affecting its activity.

This study identifies SpeMreB1 as the most active MreB isoform in Spiroplasma, which manages SpeMreB5 polymerization to drive swimming motility.

This study shows that myosin 18Aα recruits β-Pix to dendritic spines in Purkinje neurons, enhancing spine maturation through actin and myosin filament assembly.

The paper analyzes MreB sequences from Spiroplasma to determine their phylogenetic origin and sequence features, identifying key differences from Bacillus MreBs that may relate to their unique swimming mechanism.