Author

Kenny Vu

National Institutes of HealthORCID 0000-0002-9396-1120Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Approach:statistical model· 2 Assay:whole-exome sequencing· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

Nyall R. LondoniD· 3 shared Sreenivasulu Gunti· 3 shared Andrew P. LaneiD· 2 shared Farhoud FarajiiD· 2 shared Federico ComoglioiD· 2 shared Fernando Tadeu ZamunériD· 2 shared Gary L. GalliaiD· 2 shared Lisa M. RooperiD· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

Nyall R. LondonORCID

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3 papers

Recent·Most cited

GenomicsbioRxivPreprintJun 4, 2026

scRNA-seq and genomics analyses reveal key mechanisms of inverted papilloma-associated sinonasal squamous cell carcinoma malignant transformation

Tolani F. Olonisakin, Fernando Tadeu Zamunér, Kenny Vu…·Nyall R. London

This study identifies molecular and immune mechanisms driving malignant transformation from inverted papilloma to sinonasal squamous cell carcinoma using multi-omics analyses.

DOI 10.64898/2026.06.01.729345

humanscRNA-seq

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Cancer BiologyNature CommunicationsPeer-reviewedJun 11, 2025

Molecular patterns and mechanisms of tumorigenesis in HPV-associated and HPV-independent sinonasal squamous cell carcinoma

Fernando Tadeu Zamunér, Sreenivasulu Gunti, Gabriel J. Starrett…·Nyall R. London

This study identifies molecular differences between HPV-associated and HPV-independent SNSCC, showing HPV drives tumorigenesis and suggesting YAP/TAZ and PI3K inhibition as therapeutic targets.

11 citationstop 2%

humanwhole-exome sequencing

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Cancer BiologybioRxivPreprintMay 19, 2025

In vivo CRISPR screening identifies NF1/RASA1/TP53 co-mutations and downstream MEK signaling as a common key mechanism of sinonasal tumorigenesis

Kenny Vu, Sreenivasulu Gunti, Ramya Viswanathan…·Nyall R. London

This study identifies NF1/RASA1/TP53 co-mutations and MEK signaling as key drivers of sinonasal tumorigenesis through in vivo CRISPR screening and drug response testing.

DOI 10.1101/2025.05.19.654661

mousegenetic perturbation / CRISPR

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