Author

Nyall R. London

National Institutes of HealthORCID 0000-0002-2902-4407Unclaimed ORCID profile · follow to get notified of new preprints

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Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Approach:statistical model· 2 Assay:whole-exome sequencing· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

Kenny VuiD· 3 shared Sreenivasulu Gunti· 3 shared Andrew P. LaneiD· 2 shared Farhoud FarajiiD· 2 shared Federico ComoglioiD· 2 shared Fernando Tadeu ZamunériD· 2 shared Gary L. GalliaiD· 2 shared Lisa M. RooperiD· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

Kenny VuORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

scRNA-seq and genomics analyses reveal key mechanisms of inverted papilloma-associated sinonasal squamous cell carcinoma malignant transformation

Tolani F. Olonisakin, Fernando Tadeu Zamunér, Kenny Vu, Sreenivasulu Gunti, Y M Ding, Yue Hou, Xinping Yang, Ramya Viswanathan, Angel Huynh, Federico Comoglio, Marco Notaro, Christopher Cherry, Michael Patatanian, Nathan W. Wong, Timothy R. DeKlotz, , Nyall R. London

This study identifies molecular and immune mechanisms driving malignant transformation from inverted papilloma to sinonasal squamous cell carcinoma using multi-omics analyses.

bioRxiv · Jun 4, 2026 · PreprintGenomics

Molecular patterns and mechanisms of tumorigenesis in HPV-associated and HPV-independent sinonasal squamous cell carcinoma

Fernando Tadeu Zamunér, Sreenivasulu Gunti, Gabriel J. Starrett, Farhoud Faraji, Tiffany Toni, Anirudh Saraswathula, Kenny Vu, Anuj Kumar Gupta, Yan Zhang, Daniel L. Faden, Michael E. Bryan, Theresa W. Guo, Nicholas R. Rowan, Murugappan Ramanathan, Andrew P. Lane, , Nyall R. London

This study identifies molecular differences between HPV-associated and HPV-independent SNSCC, showing HPV drives tumorigenesis and suggesting YAP/TAZ and PI3K inhibition as therapeutic targets.

Nature Communications · Jun 11, 2025Peer-reviewedCancer Biology

In vivo CRISPR screening identifies NF1/RASA1/TP53 co-mutations and downstream MEK signaling as a common key mechanism of sinonasal tumorigenesis

Kenny Vu, Sreenivasulu Gunti, Ramya Viswanathan, Anjali Nandal, Riley M. Larkin, Sung‐Woo Cho, Jonathan Zou, Shivani Ramolia, Austin T.K. Hoke, Siani Barbosa, Gary L. Gallia, Lisa M. Rooper, Charalampos S. Floudas, Hui Wen Cheng, Christine N. Miller, , Nyall R. London

This study identifies NF1/RASA1/TP53 co-mutations and MEK signaling as key drivers of sinonasal tumorigenesis through in vivo CRISPR screening and drug response testing.

bioRxiv · May 19, 2025 · PreprintCancer Biology