J. Michael Harnish

This study uses Drosophila to express Zika virus proteins and identifies phenotypes and host pathways disrupted by the virus during development.

The paper functionally tests ASD missense variants in Drosophila, finding 38% caused functional alterations and identifying GLRA2 as a cause of neurodevelopmental phenotypes.

ModelMatcher connects researchers, clinicians, and stakeholders in rare disease research to accelerate translational studies and collaborations.