Author

Erika López‐Arribillaga

Institute for Research in BiomedicineORCID 0000-0002-9451-6635Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genomics· 2 Bioinformatics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:statistical model· 2 Assay:whole-genome sequencing· 2 Assay:bulk RNA-seq· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Núria López-BigasiD· 3 shared Ailith EwingiD· 2 shared Christine FeigiD· 2 shared Claudia Arnedo-PaciD· 2 shared Colin A M SempleiD· 2 shared Craig J. AndersoniD· 2 shared Duncan T. OdomiD· 2 shared Elissavet KentepozidouiD· 2 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersGenomics

Núria López-BigasORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Genetic background sets the trajectory of cancer evolution

Sarah J. Aitken, Frances Connor, Christine Feig, Tim F. Rayner, Margus Lukk, Juliet Luft, Stuart Aitken, Claudia Arnedo-Pac, J.V. Hayes, Michael D. Nicholson, Ailith Ewing, Vasavi Sundaram, Jan C. Verburg, John Connelly, Craig J. Anderson, , Duncan T. Odom

This study shows that genetic background and environmental factors determine the trajectory of cancer evolution in mice, with distinct mutational paths despite similar final tumor states.

Nature · Jul 22, 2026Peer-reviewedGenomics

Strand-resolved mutagenicity of DNA damage and repair

Craig J. Anderson, Lana Talmane, Juliet Luft, John C. Connelly, Michael D. Nicholson, Jan C. Verburg, Oriol Pich, Susan E. Campbell, Marco Giaisi, Pei‐Chi Wei, Vasavi Sundaram, Frances Connor, Paul Adrian Ginno, Takayo Sasaki, David M. Gilbert, , Martin S. Taylor

The paper investigates how strand-asymmetric processes like replication and transcription influence DNA damage, repair, and mutagenesis, revealing mechanisms that shape cancer genome evolution.

Nature · Jun 12, 2024Peer-reviewedGenomics

Disruption of ubiquitin mediated proteolysis is a widespread mechanism of tumorigenesis

Francisco Martínez-Jiménez, Ferran Muiños, Erika López‐Arribillaga, Núria López-Bigas, Abel González-Pérez

The paper identifies degrons and E3 ligase alterations across cancer types, finding that at least 1 in 7 driver mutations affect these components.

bioRxiv · Dec 28, 2018 · PreprintBioinformatics