Author

Clayton M. Carey

University of UtahORCID 0000-0002-9458-649XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Genetics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Organism:zebrafish· 2 Research Focus:benchmarking· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

James A. GagnoniD· 2 shared Alexis V. SchmidiD· 1 shared Apurva A. GovandeiD· 1 shared Brent W. BisgroveiD· 1 shared Christopher J. YatesiD· 1 shared Hailey L. HollinsiD· 1 shared Juliane M. Cooper· 1 shared Melissa K. Hartley· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersGenomics

James A. GagnonORCID

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Author profile →

3 papers

Recent·Most cited

MIC-Drop-seq: Scalable single-cell phenotyping of mutant vertebrate embryos

Clayton M. Carey, Saba Parvez, Zachary J. Brandt, Brent W. Bisgrove, Christopher J. Yates, Randall Theodore Peterson, James A. Gagnon

MIC-Drop-seq combines CRISPR and scRNA-seq to phenotype mutant zebrafish embryos, revealing known and novel gene functions in development.

Nature Communications · Apr 1, 2026Peer-reviewedGenomics

Distinct features of the regenerating heart uncovered through comparative single-cell profiling

Clayton M. Carey, Hailey L. Hollins, Alexis V. Schmid, James A. Gagnon

This study compares cardiac injury responses in zebrafish and medaka to identify features distinguishing regenerative and non-regenerative heart responses.

Biology Open · Mar 25, 2024Peer-reviewedDevelopmental Biology

Recurrent loss-of-function mutations reveal costs to OAS1 antiviral activity in primates

Clayton M. Carey, Apurva A. Govande, Juliane M. Cooper, Melissa K. Hartley, Philip J. Kranzusch, Nels C. Elde

The paper identifies recurrent loss-of-function mutations in OAS1 across primates, showing that the cytotoxic effects of OAS1 activation can outweigh its antiviral benefits, leading to reduced catalytic activity.

Cell Host & Microbe · Jan 31, 2019Peer-reviewedGenetics