Author

Yuan Yang

University of California, Los AngelesORCID 0000-0002-9538-6369Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Yuan Yang4 papers · Institut d'Investigació Biomédica de Bellvitge

Biology Disciplines

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Biophysics· 1 Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:Drosophila· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1 Assay:protein structure· 1

Frequent Co-Authors

Juli F. FeigoniD· 3 shared Christine Stephen· 1 shared David R. BoyeriD· 1 shared David S EisenbergiD· 1 shared Jeffrey ZhangiD· 1 shared Jiahui LuiD· 1 shared Joshua L. DolinskyiD· 1 shared Justin PiiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersBiophysics

Juli F. FeigonORCID

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3 papers

Recent·Most cited

BiophysicsNature CommunicationsPeer-reviewedJan 15, 2026

HEXIM1 inter-monomer autoinhibition governs 7SK RNA binding specificity and P-TEFb inactivation

Yuan Yang, Maria Grazia Murrali, Sabrina Galvan…·Juli F. Feigon

The paper investigates how HEXIM1 autoinhibition is overcome by 7SK RNA binding to enable P-TEFb inactivation, revealing structural and functional mechanisms of 7SK RNP assembly.

4 citationstop 4%

biophysical modelingchromatin & epigenetic regulation

𝕏 PostBluesky

NeurosciencebioRxivPreprintMar 25, 2024

How short peptides can disassemble ultra-stable tau fibrils extracted from Alzheimers disease brain by a strain-relief mechanism

Ke Hou, Peng Ge, Michael R. Sawaya…·David S Eisenberg

The paper shows a D-peptide disassembles AD-tau fibrils via a strain-relief mechanism involving oppositely twisted fibrils.

4 citations

humanprotein structure

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GeneticsGeneticsPeer-reviewedNov 20, 2023

Catalytic activity of the Bin3/MEPCE methyltransferase domain is dispensable for 7SK snRNP function in Drosophila melanogaster

Ryan J. Palumbo, Yuan Yang, Juli F. Feigon, Steven D. Hanes

This study shows that catalytic activity of Bin3 is not required for 7SK snRNP function in Drosophila, as catalytic mutants can rescue all phenotypes.

3 citations

Drosophilaexperimental assay

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