Author

Hilary J. Skalski

Michigan State UniversityORCID 0000-0002-9774-4844Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:scRNA-seq· 1 Research Focus:spatial domain detection· 1

Frequent Co-Authors

Abigail Z. BennettiD· 2 shared Amelia R. Arendt· 2 shared Ronald L. ChandleriD· 2 shared Shannon K. HarkinsiD· 2 shared Amanda G. Lopez Espinosa· 1 shared Emmanuel N. PauliD· 1 shared Galen H. HostetteriD· 1 shared Genna E. MoldovaniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersPhysiology

Abigail Z. BennettORCID

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2 papers

Recent·Most cited

Endometrial Hyperplasia Risk Is Increased by High-Fat Diet Via Estrogen-Driven Stromal Fibroblast Reprogramming Toward a Pro-Fibrotic State

Hilary J. Skalski, Abigail Z. Bennett, Lauren E. Wood, Shannon K. Harkins, Amelia R. Arendt, Amanda G. Lopez Espinosa, Gregory W. Burns, Emmanuel N. Paul, Galen H. Hostetter, Katelyn Becker, Marc Wegener, Marie Adams, Jose M. Teixeira, Kin H. Lau, Ronald L. Chandler

This paper shows that a high-fat diet increases endometrial hyperplasia risk by reprogramming stromal fibroblasts to a pro-fibrotic state driven by estrogen.

bioRxiv · Mar 24, 2026 · PreprintPhysiology

Generation and characterization of a mouse model of conditional Chd4 knockout in the endometrial epithelium.

Shannon K. Harkins, Hilary J. Skalski, Abigail Z. Bennett, Laura A. Pavliscak, Nakati C. Sany, Amelia R. Arendt, Lauren Wood, Genna E. Moldovan, Ronald L. Chandler

This study generates a mouse model with conditional Chd4 knockout in the endometrial epithelium and finds no significant structural or functional changes or tumorigenesis.

PLoS ONE · Dec 19, 2025Peer-reviewedDevelopmental Biology