Ronald L. Chandler

This paper shows that a high-fat diet increases endometrial hyperplasia risk by reprogramming stromal fibroblasts to a pro-fibrotic state driven by estrogen.

This study generates a mouse model with conditional Chd4 knockout in the endometrial epithelium and finds no significant structural or functional changes or tumorigenesis.

This study shows ARID1A maintains repressive H3.3 chromatin through interactions with CHD4 and ZMYND8, suppressing genes involved in cell motility and epithelial-to-mesenchymal transition in endometriotic cells.