Abigail Z. Bennett

This paper shows that a high-fat diet increases endometrial hyperplasia risk by reprogramming stromal fibroblasts to a pro-fibrotic state driven by estrogen.

This study identifies ULK1-driven autophagy as a mechanism by which HPV+ HNC cells degrade MHC-I, promoting immune evasion and tumor growth.

This study generates a mouse model with conditional Chd4 knockout in the endometrial epithelium and finds no significant structural or functional changes or tumorigenesis.

This study investigates HMGA2 overexpression in myometrial cells, showing it induces a stem cell-like state and contributes to fibroid development through dedifferentiation and altered extracellular matrix pathways.