Author

Anna Tejchman

Jagiellonian UniversityORCID 0000-0002-9985-7228Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Małgorzata Katarzyna SobocińskaiD· 2 shared Marcin MajkaiD· 2 shared Marta KotiD· 2 shared Paula ChlebanowskaiD· 2 shared Agata MuszyńskaiD· 1 shared Agata SzlagaiD· 1 shared Anna BłasiakiD· 1 shared Bogna BadyraiD· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Małgorzata Katarzyna SobocińskaORCID

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2 papers

Recent·Most cited

Synthetic neuromelanin as a trigger of inflammation in the brain, new mouse model of Parkinson's disease

Anna Tejchman, Martyna Strzelec, Marta Kot, Paula Chlebanowska, Bogna Badyra, Małgorzata Katarzyna Sobocińska, Luigi Zecca, Marcin Majka

This study shows that synthetic neuromelanin induces inflammation and may contribute to dopaminergic neuron degeneration in a new mouse model of Parkinson's disease.

bioRxiv · Apr 28, 2023 · PreprintNeuroscience

Mitochondrial fitness influences neuronal excitability of dopaminergic neurons from patients with idiopathic form of Parkinson's disease

Paula Chlebanowska, Agata Szlaga, Anna Tejchman, Marta Kot, Paweł S. Konieczny, Klaudia Skrzypek, Agata Muszyńska, Małgorzata Katarzyna Sobocińska, Krystyna Golebiowska, Paweł P. Łabaj, Anna Błasiak, Marcin Majka

The paper investigates mitochondrial dysfunction in iPSC-derived dopaminergic neurons from idiopathic Parkinson's disease patients, finding reduced mitochondrial fitness and ATP availability linked to decreased neuronal excitability.

bioRxiv · Apr 28, 2023 · PreprintNeuroscience