Author

Jeffrey Hsu

Cleveland ClinicORCID 0000-0002-9992-4515Unclaimed ORCID profile · follow to get notified of new preprints

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2

Frequent Co-Authors

David R. Van WagoneriD· 2 shared John BarnardiD· 2 shared Jonathan D. SmithiD· 2 shared Mina K. ChungiD· 2 shared Chandrasekhar R. KothapalliiD· 1 shared Christine Schomisch MoraveciD· 1 shared Daniela Ponce‐BalbuenaiD· 1 shared Emmanuel OpokuiD· 1 shared

Activity by Year

2019-2023 · peak 1/yr

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10 top collaborators · 37 mutual co-author links

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2 shared papersGenomics

David R. Van WagonerORCID

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2 papers

Recent·Most cited

Causal SNP regulating FAM13B expression identified for the Chr. 5q31 atrial fibrillation susceptibility loc

Gregory Tchou, Daniela Ponce‐Balbuena, Nana Liu, Shamone R. Gore-Panter, Jeffrey Hsu, Fang Liu, Emmanuel Opoku, Gregory Brubaker, Sarah M. Schumacher, Christine Schomisch Moravec, John Barnard, David R. Van Wagoner, Mina K. Chung, Jonathan D. Smith

The paper identifies rs17171731 as the causal SNP regulating FAM13B expression linked to atrial fibrillation and shows its effect on cardiomyocyte gene expression and sodium current.

JACC Basic to Translational Science · Jul 27, 2023Peer-reviewedGenomics

Atrial fibrillation associated common risk variants in SYNE2 lead to lower expression of nesprin-2α1 and increased nuclear stiffness

Nana Liu, Jeffrey Hsu, Gautam Mahajan, Han Sun, John Barnard, David R. Van Wagoner, Chandrasekhar R. Kothapalli, Mina K. Chung, Jonathan D. Smith

This study identifies AF-associated SNPs in SYNE2 that reduce SYNE21 expression and increase nuclear stiffness, potentially contributing to AF pathogenesis.

bioRxiv · Aug 5, 2019 · PreprintGenomics