Author

Cristina Colomer‐Winter

University of GenevaORCID 0000-0003-0103-4977Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:human· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Kimberly A. KlineiD· 3 shared Alexandre PersatiD· 1 shared Amanzhol KurmasheviD· 1 shared Amaury Cazenave‐GassiotiD· 1 shared Andreas R. HierlemanniD· 1 shared Annelies Sophie ZinkernageliD· 1 shared Barbara Katharina HasseiD· 1 shared Caleb M. AndersoniD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

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3 shared papersMicrobiology

Kimberly A. KlineORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

MicrobiologyProceedings of the National Academy of SciencesPeer-reviewedApr 21, 2026

A two-component system signaling hub controls enterococcal membrane remodeling in response to daptomycin

Cristina Colomer‐Winter, Zeus Jaren Nair, Jerome Chua…·Kimberly A. Kline

This study identifies a two-component system signaling hub that controls membrane remodeling in Enterococcus faecalis in response to daptomycin, linking lipid changes to antibiotic resistance.

1 citation

bacteria (other)proteomics

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MicrobiologymSystemsPeer-reviewedMar 10, 2026

Adaptation of Enterococcus faecalis to intestinal mucus revealed by a human colonic organoid model

Sofya Mikhaleva, Po-Long Hsiao, Amanzhol Kurmashev…·Alexandre Persat

This study uses human colonic organoids to reveal how Enterococcus faecalis adapts to intestinal mucus, identifying key fitness determinants and colonization strategies not observed in vitro or in vivo.

3 citationstop 4%

humanimaging

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MicrobiologyNature CommunicationsPeer-reviewedJan 14, 2026

Fsr quorum sensing system restricts biofilm growth and activates inflammation in enterococcal infective endocarditis

Haris Antypas, Verena Schmidtchen, Willy I. Staiger…·Kimberly A. Kline

This study identifies the Fsr quorum sensing system in Enterococcus faecalis as a negative regulator of biofilm growth and inflammation in infective endocarditis.

8 citationstop 1%

humanmouse

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