Author

Naoya Yamada

Helmholtz MunichORCID 0000-0003-0111-028XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Junya ItoiD· 2 shared Kiyotaka NakagawaiD· 2 shared Adam WahidaiD· 1 shared André MourãoiD· 1 shared Bettina PronethiD· 1 shared Carsten BerndtiD· 1 shared Chintogtokh BaatarjaviD· 1 shared Daisuke YamamuroiD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

Junya ItoORCID

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2 papers

Recent·Most cited

Cell BiologyMolecular CellPeer-reviewedNov 14, 2024

PRDX6 dictates ferroptosis sensitivity by directing cellular selenium mobilization

Junya Ito, Toshitaka Nakamura, Takashi Toyama…·Marcus Conrad

The paper shows PRDX6 influences ferroptosis sensitivity by managing cellular selenium levels, which affects GPX4 function.

104 citationstop 1%

humanproteomics

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Molecular BiologybioRxivPreprintJun 15, 2022

DHCR7 as a novel regulator of ferroptosis in hepatocytes

Naoya Yamada, Tadayoshi Karasawa, Takanori Komada…·Masafumi Takahashi

This study identifies DHCR7 as a regulator of ferroptosis in hepatocytes, showing that its inhibition suppresses ferroptosis through 7-DHC accumulation and highlights its therapeutic potential for liver diseases.

8 citations

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