Author

Keizō Tomonaga

Kyoto UniversityORCID 0000-0003-0405-7103Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:chromatin & epigenetic regulation· 1 Assay:cryo-EM· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1 Organism:virus· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Takehiro KandaiD· 2 shared Akiko MakinoiD· 1 shared Dennis RubbenstrothiD· 1 shared Dirk W. HöperiD· 1 shared Hiromichi MatsugoiD· 1 shared Martin BeeriD· 1 shared Masayuki HorieiD· 1 shared Modoka Sakai· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersMicrobiology

Takehiro KandaORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MicrobiologyScience AdvancesPeer-reviewedApr 10, 2026

Structure and assembly of Borna disease virus 1 nucleoprotein-RNA complexes

Yukihiko Sugita, Yuya Hirai, Shinya H. Goto…·Masayuki Horie

This study determines the structure of BoDV-1 nucleoprotein-RNA complexes using cryo-EM, revealing RNA-independent assembly mechanisms and conserved features across the Bornaviridae family.

3 citationstop 2%

viruscryo-EM

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MicrobiologyMicrobiology and ImmunologyPeer-reviewedSep 23, 2024

A single amino acid substitution in the Borna disease virus glycoprotein enhances the infectivity titer of vesicular stomatitis virus pseudotyped virus by altering membrane fusion activity

Yusa Akiba, Hiromichi Matsugo, Takehiro Kanda…·Keizō Tomonaga

DOI 10.1111/1348-0421.13172

experimental assayhost-pathogen & immune response

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MicrobiologybioRxivPreprintApr 15, 2024

The absence of superinfection exclusion of Borna disease virus 2 maintains genomic polymorphisms in persistently infected cells

Takehiro Kanda, Pauline Dianne Santos, Dirk W. Höper…·Keizō Tomonaga

This study identifies genetic variations in BoDV-2 that enhance its growth and reveals its ability to maintain genomic polymorphisms in infected cells by lacking superinfection exclusion.

DOI 10.1101/2024.04.15.589485

mousewhole-genome sequencing

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