Author

Mariana Gomes da Silva Araujo

Centre international de recherche sur le cancerORCID 0000-0003-0484-5505Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Akram GhantousiD· 1 shared Aurèlie Sallé· 1 shared Caroline P. PoubeliD· 1 shared Christine CarreiraiD· 1 shared Claire RenardiD· 1 shared Cyrille Cuenin· 1 shared Ekaterina Bourova-FliniD· 1 shared Erika C. CossetiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Akram GhantousORCID

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1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintDec 12, 2024

Disruption of the epigenetic regulator BAP1 drives chromatin remodeling leading to the emergence of cells with breast cancer stem cell properties and aberrant glycosylation.

Mariana Gomes da Silva Araujo, Aurèlie Sallé, Vincent Cahais…·Rita Khoueiry

This study shows that disrupting BAP1, an epigenetic regulator, causes chromatin remodeling and leads to breast cancer stem cell-like properties and altered glycosylation.

DOI 10.1101/2024.12.12.628129

humanproteomics

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