Author

Akram Ghantous

Centre international de recherche sur le cancerORCID 0000-0002-2582-6402Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Organism:human· 2 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1

Frequent Co-Authors

Claire RenardiD· 2 shared Cyrille Cuenin· 2 shared Stéphane Keïta· 2 shared Vincent CahaisiD· 2 shared Zdenko HercegiD· 2 shared Assunta VenutiiD· 1 shared Aurèlie Sallé· 1 shared Caroline P. PoubeliD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

Claire RenardORCID

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2 papers

Recent·Most cited

Molecular and cell phenotype programs in oral epithelial cells directed by co-exposure to arsenic and smokeless tobacco

Samrat Das, Shefali Thakur, Vincent Cahais, François Virard, Liesel Claeys, Claire Renard, Cyrille Cuenin, Marie‐Pierre Cros, Stéphane Keïta, Assunta Venuti, Cécilia Sirand, Akram Ghantous, Zdenko Herceg, Michael Korenjak, Jiří Zavadil

This study investigates the molecular and cellular responses of oral epithelial cells to arsenic and smokeless tobacco exposure using multi-omics analysis and identifies key mechanisms linked to cancer progression.

BioFactors · Mar 1, 2025Peer-reviewedCancer Biology

Disruption of the epigenetic regulator BAP1 drives chromatin remodeling leading to the emergence of cells with breast cancer stem cell properties and aberrant glycosylation.

Mariana Gomes da Silva Araujo, Aurèlie Sallé, Vincent Cahais, Claire Renard, Cyrille Cuenin, Caroline P. Poubel, Stéphane Keïta, Thorsten Mosler, Christine Carreira, Gabrielle Goldman Levy, Lya Parres, Ekaterina Bourova-Flin, Sophie Rousseaux, Saadi Khochbin, Akram Ghantous, , Rita Khoueiry

This study shows that disrupting BAP1, an epigenetic regulator, causes chromatin remodeling and leads to breast cancer stem cell-like properties and altered glycosylation.

bioRxiv · Dec 12, 2024 · PreprintCancer Biology