Author

Emily R. Ko

Duke UniversityORCID 0000-0003-0542-3750Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Genetic & Genomic Medicine· 1 Genetics· 1 Genomics· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Approach:statistical model· 3 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Assay:ATAC-seq· 1 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:database / resource· 1

Frequent Co-Authors

Christopher W. WoodsiD· 3 shared Micah T. McClainiD· 3 shared Thomas W. BurkeiD· 3 shared Dennis C. KoiD· 2 shared Ephraim L. TsalikiD· 2 shared Geoffrey S. GinsburgiD· 2 shared Liuyang WangiD· 2 shared Ricardo HenaoiD· 2 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersGenetics

Christopher W. WoodsORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

GeneticsCell GenomicsPeer-reviewedNov 1, 2022

Single-cell genome-wide association reveals a nonsynonymous variant in ERAP1 confers increased susceptibility to influenza virus

Benjamin H. Schott, Liuyang Wang, Xinyu Zhu…·Dennis C. Ko

The paper identifies a nonsynonymous ERAP1 variant associated with increased susceptibility to influenza virus using scHi-HOST, a single-cell method for genetic susceptibility testing.

12 citations

humanscRNA-seq

𝕏 PostBluesky

Genetic & Genomic MedicineGenome MedicinePeer-reviewedMay 17, 2021

An atlas connecting shared genetic architecture of human diseases and molecular phenotypes provides insight into COVID-19 susceptibility

Liuyang Wang, Thomas J. Balmat, Alejandro L. Antonia…·Dennis C. Ko

The paper connects genetic architecture of human diseases with molecular phenotypes to uncover mechanisms of COVID-19 susceptibility.

86 citationstop 1%

humanGWAS summary statistics

𝕏 PostBluesky

GenomicsbioRxivPreprintDec 4, 2020

Chromatin remodeling in peripheral blood cells reflects COVID-19 symptom severity

Nicholas S. Giroux, Shengli Ding, Micah T. McClain…·Xiling Shen

Chromatin accessibility in immune cells from COVID-19 subjects is remodeled prior to seroconversion to reflect disease severity.

9 citations

humanATAC-seq

𝕏 PostBluesky