Tenghuan Ge

This study uses a mouse-adapted SARS-CoV-2 strain to model long-term post-acute sequelae of COVID-19, finding persistent neurological and multi-organ impairments in mice after mild infection.

This study shows SARS-CoV-2 causes blood-brain barrier and choroid plexus barrier damage and vascular inflammation in mice, contributing to neurological complications.

The paper identifies Atp13a5 as a specific marker for CNS pericytes in mice and develops a transgenic model to study their biology.

This study shows that microvascular injury from mild TBI accelerates Alzheimer-like pathology in mice, suggesting vascular repair could mitigate AD risk.