Kai Chang

This study shows FOXO maintains neuromuscular junction integrity during aging in Drosophila by repressing MAPK signaling.

This study shows that mTORC2 protects Drosophila hearts from HFD-induced mitochondrial and cardiac dysfunction by maintaining mitochondrial homeostasis.

This study shows activin signaling negatively regulates cardiac autophagy and health during aging in Drosophila, with mTORC2 playing a key role.

This study identifies a physical and genetic interaction between dFOXO and Kr-h1 in Drosophila, showing that Kr-h1 represses lipolysis by regulating adipose lipase brummer transcription.