Anja Lührmann

This study shows that Coxiella burnetii forms a SCV-like persistent form under hypoxia, enhancing its survival and infectivity.

This study identifies TFEB and MCOLN1 as critical for Coxiella burnetii egress through lysosomal exocytosis, showing their role in CCV development and membrane damage.

This paper shows that Coxiella burnetii uses its lipid A acylation pattern to avoid activating the non-canonical inflammasome in macrophages, thereby evading immune detection and clearance.