Susan H. Griffin

This study identifies MEIS1 as an activator of myocardin and vascular smooth muscle cell differentiation, showing its role in promoting the quiescent, differentiated state.

This study shows that loss of Leiomodin1 in vascular smooth muscle cells causes rapid coronary atherosclerosis in mice, independent of actin nucleation, and highlights Thbs1 as a target for mitigating foam cell formation.

This paper compares two CreERT2 mice, showing Itga8-CreERT2 avoids a lethal intestinal phenotype seen with Myh11-CreERT2 when knocking out Srf.

CRISPR-LRS maps transgene integration loci and estimates copy numbers in mice, aiding breeding and phenotyping.