Author

Austin Bell‐Hensley

National Institutes of HealthORCID 0000-0003-1498-3012Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Abdull J. MassriiD· 2 shared Francesco John DeMayoiD· 2 shared MyeongJin YiiD· 2 shared Shuyun LiiD· 2 shared Skylar Montague RedeckeiD· 2 shared Akshadha Jain· 1 shared Alyssa EricksoniD· 1 shared Anne Marie Z. JukiciD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersMolecular Biology

Abdull J. MassriORCID

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3 papers

Recent·Most cited

Molecular BiologyPharmacological ResearchPeer-reviewedMar 17, 2026

Impact of vitamin D deficiency on defective endometrial decidualization and the repressive role of vitamin D receptor (VDR) in the epigenomic network

MyeongJin Yi, Skylar Montague Redecke, Tianyuan Wang…·Francesco John DeMayo

This study shows that vitamin D receptor (VDR) regulates endometrial decidualization by modulating chromatin accessibility and gene expression, with implications for fertility and uterine disorders.

2 citations

mousebulk RNA-seq

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Molecular BiologyF&S SciencePeer-reviewedAug 11, 2025

Deciphering ESR1-driven transcription in human endometrial stromal cells via transcriptome, cistrome, and integration with chromatin landscape

Skylar Montague Redecke, Austin Bell‐Hensley, Shuyun Li…·Francesco John DeMayo

This study investigates ESR1 and estrogen-driven transcription in human endometrial stromal cells using transcriptome, cistrome, and chromatin landscape data to identify gene regulation and its implications in decidualization and endometrial cancer.

1 citation

humanbulk RNA-seq

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NeuroscienceCell ReportsPeer-reviewedNov 1, 2023

Distinct disease mutations in DNMT3A result in a spectrum of behavioral, epigenetic, and transcriptional deficits

Diana C. Beard, Xiyun Zhang, Dennis Y. Wu…·Harrison W. Gabel

This study examines how different DNMT3A mutations cause varied behavioral, epigenetic, and transcriptional issues in neurodevelopmental disorders.

29 citationstop 3%

mouseDNA methylation

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