Claudia Antonella Colque

This study shows that antibiotic selection in a human airway tissue model of Pseudomonas aeruginosa leads to evolution of less virulent, tissue-preserving infections with enhanced survival.

This paper develops a dual-cell air-liquid interface model to study epithelial-immune interactions during airway infection, finding that macrophages modulate inflammation and bacterial distribution.

This study uses fluorescent reporters to analyze Pseudomonas aeruginosa growth activity in human airway epithelial models, revealing strain-specific colonization patterns and growth hierarchies across infection niches.

The paper investigates infection dynamics and virulence of Pseudomonas aeruginosa in human airway epithelial models, identifying four infection clusters based on strain-specific virulence and host response.