Author

Xiaomin Xing

University of IowaORCID 0000-0003-1883-0611Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:Drosophila· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1

Frequent Co-Authors

Chun‐Fang WuiD· 3 shared Tristan C. D. G. O’HarrowiD· 2 shared Atsushi UedaiD· 1 shared Atsushi Ueda· 1 shared John Robert ManakiD· 1 shared Salleh N. EhaidebiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 12 mutual co-author links

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3 shared papersNeuroscience

Chun‐Fang WuORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintAug 25, 2024

Simultaneous GCaMP imaging and focal recording of tonic and phasic synapses: Probing short-term plasticity within a defined microenvironment

Xiaomin Xing, Chun‐Fang Wu

The paper develops a protocol for simultaneous GCaMP imaging and focal recording in Drosophila neuromuscular junctions to study short-term synaptic plasticity dynamics.

1 citation

Drosophilaimaging

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NeurosciencebioRxivPreprintDec 31, 2021

Abnormal larval neuromuscular junction morphology and physiology in Drosophila Prickle isoform mutants with defective axonal transport and adult seizure behavior

Tristan C. D. G. O’Harrow, Atsushi Ueda, Xiaomin Xing…·Chun‐Fang Wu

The paper investigates Drosophila Prickle isoform mutants, revealing structural and functional defects in larval neuromuscular junctions and adult seizure behavior.

DOI 10.1101/2021.12.31.474668

Drosophilaelectronic health records

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NeurosciencebioRxivPreprintNov 27, 2020

Investigations into modifications of neuromuscular physiology by axonal transport disruptions in Drosophila SOD1 mutants

Tristan C. D. G. O’Harrow, Atsushi Ueda, Xiaomin Xing, Chun‐Fang Wu

This study investigates how mutations in axonal transport genes modify neuromuscular defects in Drosophila SOD1 mutants, revealing isoform-specific effects on survival and junction morphology.

DOI 10.1101/2020.11.27.399956

Drosophilaexperimental assay

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