Chun‐Fang Wu

This study examines how oxidative stress and aging affect neuromuscular function in Drosophila by analyzing nerve conduction, synaptic transmission, and muscle excitability in a male-specific abdominal neuromuscular junction.

This study investigates how genetic vitamin B6 deficiency interacts with alcohol consumption in Drosophila, showing altered behavior and metabolism, with potential therapeutic implications.

The paper examines the expression and localization of two opposing dopamine receptors in Drosophila neurons, revealing their presynaptic enrichment and starvation-dependent regulation.

The paper develops a protocol for simultaneous GCaMP imaging and focal recording in Drosophila neuromuscular junctions to study short-term synaptic plasticity dynamics.

The paper shows intense light triggers male-male courtship behavior in wild-type Drosophila, revealing visual stimulation can override normal olfactory restraint.

This study shows that PNPO variants in Drosophila interact with diet to produce diverse phenotypes, including seizures and lethality, and highlights the utility of a knock-in model for studying human PNPO deficiency.

The paper investigates Drosophila Prickle isoform mutants, revealing structural and functional defects in larval neuromuscular junctions and adult seizure behavior.

This paper describes a method to record local field potentials (LFPs) in Drosophila brains during seizures, revealing distinct LFP patterns associated with different seizure types.

This study compares the effects of Sod loss-of-function mutations and paraquat-induced oxidative stress on neuromuscular physiology in Drosophila, finding distinct impacts on neurotransmission.

This study investigates how mutations in axonal transport genes modify neuromuscular defects in Drosophila SOD1 mutants, revealing isoform-specific effects on survival and junction morphology.

This study identifies age-vulnerable motor circuit functions in Drosophila under oxidative and temperature stress, showing distinct aging patterns and highlighting potential neurophysiological markers.

This study establishes Drosophila models of PNPO deficiency, showing seizures and lethality linked to low PLP levels, and demonstrates functional conservation between human and fly PNPO.

The paper quantitatively analyzes spike patterns in Drosophila flight muscles to distinguish electroconvulsion-induced seizures from native motor patterns during flight and grooming.