Author

Laurent Turchi

Centre National de la Recherche ScientifiqueORCID 0000-0003-2020-5831Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Elias A. El-HabriD· 2 shared Hervé ChneiweissiD· 2 shared Marie‐Pierre JunieriD· 2 shared Thierry VirolleiD· 2 shared Ahmed IdbaïhiD· 1 shared Alexa Saliou· 1 shared Andreas De La Vara· 1 shared Annie LadouxiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Elias A. El-HabrORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologyCell Death and DiseasePeer-reviewedAug 5, 2026

Human cortico-vascular assembloids reveal a CELF2-AHNAK-dependent switch from neuronal to endothelial tropism in glioblastoma cells

Michele Bertacchi, Annie Ladoux, Alexa Saliou…·Thierry Virolle

This study develops a human cortico-endothelial assembloid model to investigate glioblastoma cell tropism, identifying CELF2 and AHNAK as key regulators of neuronal versus endothelial invasion.

DOI 10.1038/s41419-026-09169-z

humanscRNA-seq

𝕏 PostBluesky

Cancer BiologyCellular and Molecular Life SciencesPeer-reviewedMar 21, 2025

Nicotinamide Metabolism Reprogramming Drives Reversible Senescence of Glioblastoma Cells

Ashwin Narayanan, Mirca Saras Saurty-Seerunghen, Jessica Michieletto…·Elias A. El-Habr

This study shows nicotinamide induces reversible senescence in glioblastoma cells by reprogramming metabolism and reducing SOX2, with senescent cells retaining tumor-forming ability.

10 citationstop 3%

humanbulk RNA-seq

𝕏 PostBluesky