Anthony G. Hudetz

This study shows that thalamic LIFU affects the latency of reporting conscious visual experience, with effects varying by target location and acoustic parameters.

This study examines how anesthesia affects cortical traveling waves across different states, finding frequency-specific changes and a paradoxical state resembling wakefulness during deep anesthesia.

This study identifies four wave propagation motifs in the human brain across different states of consciousness and shows how they reconfigure in diminished and psychedelic states.

This study identifies seven cortical states during desflurane anesthesia, revealing a paradoxical activated state with increased complexity during deep anesthesia.

The paper examines integration-segregation patterns in brain networks across psychedelic and sedated states using functional MRI, finding opposing configurations in these states.

This study uses low-intensity focused ultrasound to modulate thalamic regions and shows how they affect visual perception and object recognition.

This study investigates how five psychedelic drugs affect local brain synchrony and global functional connectivity using fMRI, finding disrupted local synchrony and enhanced global connectivity across all drugs.

This study examines the effects of intravenous psilocybin and DMT on sleep-wake states and EEG functional connectivity in rats, finding short-term changes in sleep patterns and increased high gamma coherence.

The paper introduces an fMRI-based measure of integration-segregation balance to differentiate conscious and unconscious brain states, finding significant shifts during anesthesia and sleep.

The paper uses fMRI to compare neural connectivity in psychedelic, sleep, and sedated states, finding global connectivity increases in psychedelics and decreases in sleep/sedation, supporting global theories of consciousness.

This study examines how propofol affects thalamic core-matrix architecture using fMRI in humans, finding a shift toward unimodal geometry during unconsciousness.

The paper uses microstimulation to study how anesthesia affects effective connectivity in rat visual cortex, finding increased network connectivity as animals wake up.

The paper identifies that co-activation of the ventral posterolateral nucleus and global brain activity correlates with physiological arousal but not perceptual awareness.

This study shows nitrous oxide reduces functional differentiation in brain networks and correlates with psychedelic experiences.

This study examines how different levels of anesthesia affect spike sequences in the visual cortex of rats during wakefulness and under anesthesia.

This study shows that both canonical and non-canonical psychedelics induce similar changes in brain network connectivity in humans.

The paper uses a computational model to simulate how anesthesia affects neuronal networks and how cholinergic effects can reverse these changes.

The paper identifies cortical hubs associated with consciousness recovery in UWS patients and finds the anterior precuneus is a key region.

The paper identifies higher-order sensorimotor brain hubs that correlate with consciousness levels across different unconscious states using resting-state fMRI data.

The paper examines how anesthesia affects visual cortex neurons categorized by firing rate and population coupling, finding distinct response changes across groups.

This study investigates how cortical neuronal activity changes with dynamic brain states under anesthesia, revealing distinct activity patterns and a paradoxical desynchronized state in deep anesthesia.

The paper develops a neural network model linking ATP production to EEG changes under anesthesia, showing reduced ATP causes slow oscillations and burst suppression.

This study investigates how network criticality influences integrated information (Phi) in human brain networks using simulations and EEG data during different consciousness states.

The paper shows that cholinergic stimulation of the prefrontal cortex restores wakefulness in anesthetized rats, while noradrenergic stimulation does not.