Olga Valverde

This study shows that prenatal alcohol exposure disrupts circadian rhythms and gene expression in mice, affecting behavior and memory.

This study shows chronic circadian disruption in adolescent mice impairs hippocampal memory and disrupts gene expression oscillations in the hippocampus and hypothalamus.

This study shows that brief light cycle shifts disrupt molecular circadian rhythms in mice, despite minimal behavioral changes.

This study shows that Bmal1 deficiency disrupts circadian rhythms and alters reward-related gene expression, leading to increased motivation for natural rewards in mice.

This study shows that a single cocaine dose impairs eCB-LTD and social behavior in mice, which can be reversed by FAAH inhibition.

This study shows that PPAR-γ in hippocampal astrocytes can reverse memory deficits caused by early alcohol exposure in mice.

This study examines Bmal1-knockout mice, finding reduced cocaine-seeking behavior and cognitive impairments linked to altered dopamine metabolism.

This study develops a mouse model of postpartum depression and evaluates the neurobiological effects of ketamine and allopregnanolone treatments.

This study shows that cannabidiol treatment during peri-adolescence improves cognitive deficits and reduces neuroinflammation in a mouse model of fetal alcohol spectrum disorder.

This study identifies shared transcriptomic changes between Alzheimer's disease and Major Depression in mice, suggesting Major Depression may be an early manifestation of Alzheimer's.

The paper investigates how maternal separation with early weaning affects impulsivity and AMPA receptor subunit expression in male and female mice, finding sex-specific differences in glutamatergic function and cocaine-seeking behavior.

This paper develops a mouse model to study cocaine-seeking behavior and incubation of craving, finding that cannabidiol reduces cocaine self-administration but not craving incubation, depending on administration timing.

This study investigates cannabidiol's effects on cocaine seeking behavior in mice, finding it blocks cue-induced reinstatement but facilitates stress-induced reinstatement, with CB1 receptor involvement.