Inés Gallego‐Landin

This study shows that prenatal alcohol exposure disrupts circadian rhythms and gene expression in mice, affecting behavior and memory.

This study shows chronic circadian disruption in adolescent mice impairs hippocampal memory and disrupts gene expression oscillations in the hippocampus and hypothalamus.

This study shows that brief light cycle shifts disrupt molecular circadian rhythms in mice, despite minimal behavioral changes.

This study shows that Bmal1 deficiency disrupts circadian rhythms and alters reward-related gene expression, leading to increased motivation for natural rewards in mice.

This study shows that PPAR-γ in hippocampal astrocytes can reverse memory deficits caused by early alcohol exposure in mice.

This study examines Bmal1-knockout mice, finding reduced cocaine-seeking behavior and cognitive impairments linked to altered dopamine metabolism.

This study develops a mouse model of postpartum depression and evaluates the neurobiological effects of ketamine and allopregnanolone treatments.