Author

Xueyi Li

Stanford Cardiovascular InstituteORCID 0000-0003-2358-5171Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Xueyi Li1 paper · Massachusetts General Hospital

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Jinliang LiiD· 3 shared Michael S. KapiloffiD· 3 shared Ramesh V. NairiD· 2 shared Vi Bich Nguyen· 2 shared Abraham L. BayeriD· 1 shared Adam R. WendeiD· 1 shared Alessia FornoniiD· 1 shared Alexander GrabneriD· 1 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersPhysiology

Jinliang LiORCID

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3 papers

Recent·Most cited

PhysiologybioRxivPreprintJul 28, 2026

Gene therapy targeting of AKAP6β-CaMKII signalosomes improves myocardial inflammation and heart failure in a swine model of cardiometabolic syndrome

Darla L. Tharp, Sofia M. Possidento, Jinliang Li…·Michael S. Kapiloff

DOI 10.64898/2026.07.23.740435

bulk RNA-seqspatial transcriptomics

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Cell BiologybioRxivPreprintFeb 17, 2026

Regulation of the Balance between Concentric and Eccentric Cardiac Hypertrophy by a CDC14A-KMT5A Signaling Pathway

Xueyi Li, Jinliang Li, Yuliang Tan…·Michael S. Kapiloff

This paper identifies CDC14A and KMT5A as regulators of concentric vs. eccentric cardiac remodeling through epigenomic control of gene transcription, with CDC14A as a potential therapeutic target for heart failure.

DOI 10.64898/2026.02.16.706249

mouseproteomics

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Molecular BiologyScientific ReportsPeer-reviewedMay 5, 2022

FGF21-FGFR4 signaling in cardiac myocytes promotes concentric cardiac hypertrophy in mouse models of diabetes

Christopher Yanucil, Dominik Kentrup, Xueyi Li…·Christian Faul

This study shows that FGF21-FGFR4 signaling in cardiac myocytes causes concentric hypertrophy in diabetic mice, suggesting it may contribute to HFpEF and that FGFR4 blockade could be a therapeutic target.

25 citations

mouseexperimental assay

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