Joel Bruss

This study examines how domain-general cognition (g) affects lesion-deficit mapping by analyzing 473 patients with focal brain damage using structural and functional lesion network mapping.

This study uses TMS combined with iEEG and fMRI to show that parietal TMS guided by hippocampal connectivity engages and modulates hippocampal activity, with effects related to individual FC strength and theta oscillations.

This study uses TMS-iEEG to show that dlPFC stimulation evokes activity in sgACC, providing electrophysiologic evidence of its connectivity.

The paper introduces an open-source MATLAB toolkit for inferential and predictive modeling of imaging-behavior and lesion-deficit relationships, demonstrating its use in analyzing language impairments linked to brain lesions.

This study examines how focal brain damage affects voluntary respiratory and motor control, finding impaired regulation and increased anxiety in specific brain regions.

This paper improves MVAR model estimation for electrophysiological data by incorporating fMRI priors, reducing data needs and improving model accuracy.

The paper examines brain activity patterns in stroke patients, finding that increased time in a frontoparietal brain state correlates with better motor recovery in those with dominant-hand corticospinal tract damage.

This study identifies brain regions and networks associated with time disorientation in patients with focal brain lesions.

The paper maps auditory cortical networks using resting state intracranial electrophysiology, revealing hierarchical organization across spatial scales and frequency-dependent hub identities.

This study uses intracranial electrocorticography to evaluate the effects of transcranial magnetic stimulation in humans, showing localized and network-level brain responses.

This study uses graph matching to analyze functional connectivity reorganization in stroke patients, showing it correlates with motor recovery and structural disconnection.

The paper investigates thalamic hubs in the human brain and finds that lesions in highly connected subregions cause broader cognitive deficits than those in less connected regions.

The paper examines functional connectivity of surgically disconnected temporal pole using resting-state fMRI and finds significant connectivity with remote regions despite anatomical disconnection.