Author

Borja Belandia

Consejo Superior de Investigaciones CientíficasORCID 0000-0003-2448-206XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:mouse· 1

Frequent Co-Authors

Cristina Garnés‐GarcíaiD· 3 shared Francisco J. CimasiD· 3 shared María José Ruiz‐HidalgoiD· 3 shared Pablo Fernández‐ArocaiD· 3 shared Ricardo Sánchez‐PrietoiD· 3 shared Diego M. Fernández‐ArocaiD· 2 shared Jaime Jiménez‐Suárez· 2 shared Natalia García-FloresiD· 2 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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3 shared papersCancer Biology

Cristina Garnés‐GarcíaORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 15, 2026

Revisiting AXL as a therapeutic target in soft tissue sarcoma: transcriptomic profiling reveals largely AXL-independent activity of dubermatinib and bemcentinib

Cristina Garnés‐García, Pablo Fernández‐Aroca, Francisco J. Cimas…·Diego M. Fernández‐Aroca

This study shows that AXL inhibitors dubermatinib and bemcentinib have antitumour effects in soft tissue sarcoma that are largely independent of AXL inhibition.

DOI 10.64898/2026.10.02.755908

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologyInternational Journal of Biological SciencesPeer-reviewedFeb 4, 2026

VCAN is essential for ERK5-driven tumorigenesis in soft tissue sarcoma

Jaime Jiménez‐Suárez, Francisco J. Cimas, José Joaquín Paricio…·Ricardo Sánchez‐Prieto

This study identifies VCAN as a novel transcriptional target of ERK5 and shows its essential role in ERK5-driven tumorigenesis in soft tissue sarcoma.

1 citation

mousebulk RNA-seq

𝕏 PostBluesky

Cancer BiologyMolecular OncologyPeer-reviewedDec 3, 2024

The signaling axis CDK12-BRCA1 mediates dinaciclib associated radiosensitivity through p53-mediated cellular senescence.

Natalia García-Flores, Diego M. Fernández‐Aroca, Cristina Garnés‐García…·Ricardo Sánchez‐Prieto

This paper shows dinaciclib increases radiosensitivity in some cancer cell lines by inhibiting CDK12, reducing BRCA1, and promoting p53-dependent senescence.

7 citations

humanbulk RNA-seq

𝕏 PostBluesky