Thomas T. Ngo

This study shows that exposure to anti-GluN1 mAb during development causes long-term sensory-motor deficits and callosal axon changes in mice.

This study shows that Shh from mossy cells supports NSC pool maintenance after seizures and in aging by promoting neurogenesis and NSC self-renewal.

This study identifies FGF5 signaling as a key driver in SHH subgroup infantile medulloblastoma, showing its upregulation and therapeutic potential through FGFR inhibition.

The paper shows that SHH signaling modulates FGF15 expression to maintain neocortical progenitor specification during corticogenesis.