Stefanie G. Wohl

This study shows that disabling Muller glia Dicer1 preserves retinal function after injury in mice, with the most protection seen in Rlbp1-CreER-driven mice.

The paper shows that Dicer/miRNAs in late retinal progenitor cells are essential for proper postnatal retinal development and function.

This study investigates the sequence of pathological events caused by Dicer loss in Muller glia, showing cone dysfunction precedes rod degeneration and retinal remodeling.

The paper identifies loss of neurogenic gene expression and motif accessibility in mature Muller glia that limits their regenerative capacity.