Steven Bergmans

The paper identifies an interferon-responsive oligodendroglial state in aging white matter using killifish models, linking it to potential neurodegenerative disease mechanisms.

The paper shows that dendritic pruning, linked to suppressed mTORC1 signaling, supports axonal regeneration in zebrafish, revealing a trade-off between dendritic preservation and axonal growth.

The paper compares retinal ganglion cell resilience to optic nerve injury across mice, zebrafish, and killifish, revealing species-specific differences in RGC density and injury response.

The paper identifies metabolic reprogramming in zebrafish axonal regeneration and shows its conservation in mammals, suggesting therapeutic potential for CNS regeneration.

The paper shows that local glycolysis supports axonal regeneration after injury by providing ATP, complementing mitochondrial metabolism.

This study investigates the role of oligodendroglia in Wolfram syndrome using mouse models and iPSC-derived cells, finding preserved oligodendroglia function and suggesting alternative targets for treatment.

This study uses bulk and single-cell RNA-seq to analyze age-related transcriptome changes in the African turquoise killifish retina, identifying oxidative stress, gliosis, and inflammageing, and highlighting dysregulation of retinal disease genes.