Nina R. Salama

This study investigates the fitness benefits and costs of the H. pylori Cag-T4SS and CagA during stomach infection, showing initial benefits and later costs due to inflammation.

This study identifies allelic variation in H. pylori cell surface genes that influence exoproteome binding and gastric tissue adherence through structural changes in lipopolysaccharide.

This study shows that H. pylori-induced gastric metaplasia enables F. animalis colonization, which is associated with increased Gal-GalNAc and reduced acidity.

This study identifies Muc6-expressing isthmus progenitors as key contributors to gastric regeneration and metaplasia through myeloid-mesenchymal interactions.