Author

Soledad Ochoa

The University of Texas at San Antonio Health Science CenterORCID 0000-0003-2819-0486Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Fernanda Soares Rasquel-OliveiraiD· 2 shared Kate LawrensoniD· 2 shared Michael S. RogersiD· 2 shared Anne E. Porter· 1 shared Brett David McKinnoniD· 1 shared Caroline E. GargettiD· 1 shared Cynthia KohliD· 1 shared Eva Graf· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Fernanda Soares Rasquel-OliveiraORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintOct 20, 2025

Pro-endometriosis macrophage release of IL-33 is key for endometriosis pain and lesion formation

Victor Fattori, Fernanda Soares Rasquel-Oliveira, Soledad Ochoa…·Michael S. Rogers

This study identifies IL-33 as a key mediator in endometriosis pain and lesion formation through neuroimmune communication involving CGRP and macrophages.

2 citations

mousehuman

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ImmunologyAdvanced SciencePeer-reviewedSep 12, 2025

M2 Macrophages are Major Mediators of Germline Risk of Endometriosis and Explain Pleiotropy with Comorbid Traits

Soledad Ochoa, Fernanda Soares Rasquel-Oliveira, Brett David McKinnon…·Kate Lawrenson

This study identifies M2 macrophages and IL1 signaling as key mediators of endometriosis risk and shows their pleiotropic role in comorbid traits.

10 citationstop 2%

humanbulk RNA-seq

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