Author

Jana Talbot

University of TasmaniaORCID 0000-0003-2863-4139Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Assay:proteomics· 2 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Alex William HewittiD· 2 shared Anthony L. CookiD· 2 shared Sueanne Chear· 2 shared Aidan David BindoffiD· 1 shared Alexandra GrubmaniD· 1 shared Alice PébayiD· 1 shared Anna Elizabeth KingiD· 1 shared Cheyenne Reuben-ThomasiD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCell Biology

Alex William HewittORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cell BiologyInvestigative Ophthalmology & Visual SciencePeer-reviewedNov 13, 2024

Genetic and cellular basis of impaired phagocytosis and photoreceptor degeneration in CLN3 disease.

Jimin Han, Sueanne Chear, Jana Talbot…·Ruchira Singh

This study investigates how CLN3 mutations impair phagocytosis and cause photoreceptor degeneration in CLN3 disease using patient-derived cells and a miniswine model.

6 citations

mouseproteomics

𝕏 PostBluesky

NeuroscienceDisease Models & MechanismsPeer-reviewedDec 1, 2022

Lysosomal alterations and decreased electrophysiological activity in CLN3 disease (966 bp deletion, E295K) patient-derived cortical neurons

Sueanne Chear, Sharn E. Perry, Richard Wilson…·Anthony L Cook

This study examines CLN3 disease using patient-derived cortical neurons, finding reduced electrophysiological activity and lysosomal alterations.

18 citations

humanproteomics

𝕏 PostBluesky