Author

Sharn E. Perry

University of TasmaniaORCID 0000-0002-3687-7803Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Anna Elizabeth KingiD· 2 shared Anthony L CookiD· 2 shared Aidan David BindoffiD· 1 shared Alex William HewittiD· 1 shared Alexandra GrubmaniD· 1 shared Alice PébayiD· 1 shared Andrew J PhippsiD· 1 shared Anisuzzaman ChowdhuryiD· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Anna Elizabeth KingORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

HDAC6 inhibition as a mechanism to prevent axon degeneration in the mSOD1G93A mouse model of ALS.

Andrew J Phipps, Samuel Dwyer, Jessica Marie Collins, Fariha Kabir, Rachel A.K. Atkinson, Anisuzzaman Chowdhury, Lyzette Matthews, Deepika Dixit, R. S. TERRY, Jason A. Smith, Nuri Gueven, William R. Bennett, Anthony L Cook, Anna Elizabeth King, Sharn E. Perry

This study shows HDAC6 inhibition with ACY-738 stabilizes microtubules and reduces lower motor neuron degeneration in mSOD1G93A mice, but does not prevent motor function decline.

bioRxiv · Aug 28, 2023 · PreprintNeuroscience

Lysosomal alterations and decreased electrophysiological activity in CLN3 disease (966 bp deletion, E295K) patient-derived cortical neurons

Sueanne Chear, Sharn E. Perry, Richard Wilson, Aidan David Bindoff, Jana Talbot, Tyson L. Ware, Alexandra Grubman, James Clement Vickers, Alice Pébay, Jonathan B. Ruddle, Anna Elizabeth King, Alex William Hewitt, Anthony L Cook

This study examines CLN3 disease using patient-derived cortical neurons, finding reduced electrophysiological activity and lysosomal alterations.

Disease Models & Mechanisms · Dec 1, 2022Peer-reviewedNeuroscience

Loss of Wt1 in the murine spinal cord alters interneuron composition and locomotion

Danny Schnerwitzki, Sharn E. Perry, Anna Ivanova, Fábio Viegas Caixeta, Paul Cramer, Sven Günther, Kathrin Weber, Atieh S. Tafreshiha, Lore Becker, Ingrid Liliana Vargas Panesso, Thomas Klopstock, Martin Hrabě de Angelis, Manuela Schmidt, Klas Kullander, Christoph Englert

This study shows that Wt1 deletion in mice leads to altered interneuron composition and locomotion defects.

Life Science Alliance · Aug 1, 2018Peer-reviewedNeuroscience