Author

Iskra Katic

Friedrich Miescher InstituteORCID 0000-0003-3703-5939Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Approach:experimental assay· 2 Assay:long-read sequencing· 1

Frequent Co-Authors

Collin Y. EwaldiD· 1 shared Dimos GaidatzisiD· 1 shared Helge GroßhansiD· 1 shared Panagiotis PapasaikasiD· 1 shared Rafal CioskiD· 1 shared Richard Venz· 1 shared Tina PekečiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 9 mutual co-author links

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1 shared paperMolecular Biology

Collin Y. EwaldORCID

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2 papers

Recent·Most cited

GeneticsbioRxivPreprintMay 5, 2026

Large transgene arrays cause aberrant transcription and synthetic molting defects with myrf-1 mutation

Iskra Katic, Panagiotis Papasaikas, Dimos Gaidatzis, Helge Großhans

The paper shows that large transgene arrays in C. elegans cause transcriptional dysregulation and synthetic molting defects when combined with a myrf-1 mutation.

DOI 10.64898/2026.04.30.721998

C. eleganslong-read sequencing

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Molecular BiologyeLifePeer-reviewedSep 10, 2021

End-of-life targeted auxin-mediated degradation of DAF-2 Insulin/IGF-1 receptor promotes longevity free from growth-related pathologies

Richard Venz, Tina Pekeč, Iskra Katic…·Collin Y. Ewald

This study shows that late-life targeted degradation of the DAF-2 receptor in C. elegans extends lifespan without growth-related pathologies.

66 citations

C. elegansexperimental assay

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